That's what I said over a year ago
Obamot wrote: Go and understand how incompetent people (who claim to communicate in the name of “science" s'pa) base their arguments on a political narrative when it comes to facts!
[Izentrop...! blah blah blah]
It's crazy, we can tell them that the mRNA that codes for the spike protein will damage the vascular endothelium, nothing helps! Still new scientific observations prove it, supporting evidence and experiments NOT TO HAVE THEM BY PFIZER-GLAXO, ASTRA-ZENEKA, MODERNA (etc)
This is what I have been saying here for several months, under the usual taunts. Then always from the same instituteIt is not difficult to understand then, that mRNA vaccines in people with already weakened vascular systems, will develop vascular lesions with latent effects - all over the body, whether due to the spike proteins of mutants or those vaccines - which will worsen with age and may lead to all kinds of pathologies or even early death.NEW CORONAVIRUS SPIKE PROTEIN PLAYS AN ADDITIONAL KEY ROLE IN DISEASE
Salk researchers and their collaborators show how the protein damages cells, confirming that COVID-19 is a predominantly vascular disease.
New coronavirus spike protein plays additional key role in disease
Salk researchers and their collaborators show how the protein damages cells, confirming that COVID-19 is a predominantly vascular disease.
LA JOLLA - Scientists have known for some time that the "spike" proteins characteristic of SARS-CoV-2 help the virus infect its host by attaching itself to healthy cells. Today, a major new study shows that the virus spike proteins [...] also play a key role in the disease itself. [...]
- "A lot of people think it's a respiratory disease, but it's actually a vascular disease", explains assistant research professor Uri Manor, who is the study's co-lead author. "This could explain why some people have strokes and why some people have problems in other parts of the body. What these diseases have in common is that they all have a vascular basis. "
Salk researchers collaborated with scientists at the University of California at San Diego to write this article, including lead co-author Jiao Zhang and lead co-author John Shyy.
While the results in themselves are not entirely a surprise, the article provides clear confirmation and a detailed explanation of the mechanism by which the protein damages vascular cells for the first time. There is a growing consensus that SARS-CoV-2 affects the vascular system, but it was not clear exactly how it does it. Likewise, scientists studying other coronaviruses have long suspected that the spike protein helps damage vascular endothelial cells, but this is the first time this process has been documented.
In the new study, the researchers created a "pseudovirus" that was surrounded by the classic SARS-CoV-2 spike protein crown, but which contained no real virus. Exposure to this pseudovirus resulted in damage to the lungs and arteries of an animal model, proving that the spike protein alone is sufficient to cause disease. Tissue samples revealed inflammation of the endothelial cells that line the walls of the pulmonary arteries.
The team then replicated this process in the lab, exposing healthy endothelial cells (which line the arteries) to the spike protein. They showed that the spike protein damaged cells by binding to ACE2. This binding disrupted the molecular signaling of ACE2 to mitochondria (organelles that produce energy for cells), which damaged and fragmented the mitochondria.
Previous studies have shown a similar effect when cells are exposed to the SARS-CoV-2 virus, but this is the first study to show that damage occurs when cells are exposed to S protein alone.
"If we suppress the replication capacity of the virus, it still has a major damaging effect on vascular cells, simply because of its ability to bind to this ACE2 receptor, the S protein receptor, now famous for to COVID, "Manor explains." Further studies with mutant spike proteins will also provide insight into the infectivity and severity of mutant SARS CoV-2 viruses."
The researchers then hope to take a closer look at the mechanism by which the disrupted ACE2 protein damages mitochondria and causes them to change shape.
The other authors of the study are Yuyang Lei and Zu-Yi Yuan from Jiaotong University in Xi'an, China; Cara R. Schiavon, Leonardo Andrade and Gerald S. Shadel de Salk; Ming He, Hui Shen, Yichi Zhang, Yoshitake Cho, Mark Hepokoski, Jason X.-J. Yuan, Atul Malcolm and J.-J. Yuan. J. Yuan, Atul Malhotra, Jin Zhang of University of California San Diego; Lili Chen, Qian Yin, Ting Lei, Hongliang Wang, and Shengpeng Wang from Xi'an Jiatong University Health Sciences Center in Xi'an, China.
This research was supported by the National Institutes of Health, the National Natural Science Foundation of China, the Shaanxi Natural Science Fund, the National Key Research and Development Program, the First Affiliated Hospital of Xi'an Jiaotong University and Xi 'University. an Jiaotong.
https://www.salk.edu/news-release/the-n ... n-illness/
Critics of these observations started by saying that injected into the muscle, the mRNA would go no further, only to recognize that the mRNAs would end up in the blood (and would damage the first organ in the circuit to come: as per examples the vessels of the liver (which is extremely serious. A real micro-biological time bomb).
So if you want this forum it's like the myth of Sisyphus, you have to repeat over and over again the same relevant information which is then denied. It's exhausting, and in addition we are banned and treated as parasites. Which after a year cuts off any desire to post here. And this because the forum is administered SOMETIMES very well, but unevenly, sometimes also revealing very disappointing reactions, even suggesting a little stupidity or malice.
Here is part of the riddle solved (which apparently confirms facts and hypotheses)
it is always 
