Obamot wrote:VetusLignum wrote:Christophe wrote:Interesting but the second is only based on computer simulations ... So these may be clues but not evidence (proof that we may never find ...)
Yes, we have only hypotheses for the moment.
Note however that if there had been an asymptomatic strain (ancestor) of SARS-CoV2, I think it would still be circulating, and that this would have been identified, if only because of the numerous tests of SARS -CoV2 performed worldwide in recent months.
Another hypothesis raised in the second article, which I did not mention because I do not believe in it, is that the affinity for hACE2 receptors is not sufficient to infect humans, and that another component genetics allowing the infection would have appeared recently. I don't believe it, because
1) I tell myself that if there was another factor, other researchers would have identified it
2) The affinity for hACE2 receptors would then have been useless for many years from an evolutionary point of view, and therefore, I do not see why it would have been transmitted.
Admitting this hypothesis (very interesting from the ancestor of the virus that infected bats and not the other way around) how could it be that the phylogenetic tree did not provide information on this point, since until then researchers seemed unanimous => Chinese virus => fallacy of the start of the epidemic from China: all this shatters ...!and it's not to displease me ...
Could it be that researchers have manipulated one of these ancestors, without even imagining how dangerous they were (or conversely, would have precisely manipulated this virus since knowing that this would constitute a perfect concealment as to its real origin? ... => in order to create a “demographic weapon” ...)? We are not far from the initial certainties!
It seems to me less and less obvious that research on this type of virus (which is known and which exists) would be purely fortuitous and of an interest exclusively of “fundamental research” (without any ulterior motive ...)
This hypothesis does not explain either the presence of HIV strands concentrated in the same segment, nor the fact that it gave the virus an exceptional potential for contamination, other than by the fact that the virus was manipulated (unless 'an incredible bad luck and statistically / temporally very improbable ...).
On the other hand, what strongly supports this hypothesis is the fact that the pandemic started at a time when the bats are in hibernation, which makes the “western hypothesis” of an initial outbreak in Wuhan very suspect, as soon as departure! Aside from being out of the P4 lab co-funded by the USA, I never believed it ...
Already, for me, the easiest hypothesis to justify is that SARS-CoV2 is a virus transformed in a laboratory.
Now, it remains interesting to ask what scenarios could found the alternative hypothesis, natural evolution.
For the moment, the main questions revolve around the affinity for the hACE2 receptors, which allows human infection, and which is not found in any known bat coronavirus.
According to Montagnier, it comes from the AIDS virus, and it was introduced in the laboratory.
Others say it comes from a pangolin coronavirus, which would have naturally recombined with a bat virus, producing SARS-CoV2. But this scenario is very hypothetical, in particular because we have no intermediate forms. This introduction could very well have been done in the laboratory.
And then there is the hypothesis that this affinity was already present in certain bat viruses of the Tongguan mine, and that such a bat virus capable of infecting humans would already circulate in humans. without giving symptoms, before having evolved into the symptomatic form that we are dealing with today. Again, there are no intermediate forms. It would be easy to go back to the mine and analyze everything there.
All this is very confusing, and we understand why the official version is still that the infection comes from the Huanan market: there is currently no alternative theory that holds water if we want evolution to have been natural.
However, if he had to know that SARS-CoV2 was done in the laboratory, then no one would want to pay for a vaccine; we would ask the laboratory to finance it; whereas in the case of natural evolution, there is plenty of money to be made. Hence the importance for NIH and China to do everything they can to favor the hypothesis of natural evolution.
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